By Christophe Faure, Nikhil Thapar, Carlo Di Lorenzo
This quantity offers a complete and updated theoretical evaluation and functional consultant on pediatric gastrointestinal motility and useful issues. the newest version comprises widely revised and new chapters to mirror the speedily becoming box of pediatric neurogastroenterology. New themes lined contain neurobiology of discomfort in young ones, practical oropharyngoesophageal review, dysautonomia, and psychotropic medicinal drugs. The textual content additionally positive aspects instructive illustrations, photos, and tables.
Written through world-renown specialists within the box, Pediatric Neurogastroenterology: Gastrointestinal Motility and practical issues in youngsters, moment Edition is a worthy source for pediatric gastroenterologists, grownup gastroenterologists, pediatricians, and all pros thinking about the therapy and administration of kids with such disorders.
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Additional info for Pediatric Neurogastroenterology: Gastrointestinal Motility and Functional Disorders in Children
Cells behind the wavefront begin progressive differentiation towards neural and glial cell fates. 5, GDNF and EDN3 are expressed in the distal hindgut (not shown). (From Heanue and Pachnis (2007) Enteric nervous system development and Hirschsprung’s disease: advances in genetic and stem cell studies. Nat Rev Neurosci 8(6): 466–479. , from Durbec et al. (1996) Development 122(1): 349–358) has a significant impact on the capacity of ENCCs to completely colonise the gut. In a number of experimental conditions in which the initial pool of ENCCs is reduced, there is a failure of ENCC to colonise the distal gut [21, 39, 60] or to appropriately populate the entire gut with ENS neurons .
Burns small intestine. However, closer examination revealed that ICC had not undergone apoptosis, but had developed ultrastructural features similar to smooth muscle cells. These findings highlight plasticity between ICC and smooth muscle cells that is regulated by Kit signalling [122, 123]. In addition to transdifferentiation, ICC appear to have some capacity for regeneration. In experiments where the mouse intestine was exposed to a chemical insult which induced loss of the myenteric plexus associated ICC, a few weeks later, cells with ICC-like features began to reappear [124, 125].
World J Gastroenterol. 2008;14:2953–61. 17 24. Mao J, Kim BM, Rajurkar M, et al. Hedgehog signaling controls mesenchymal growth in the developing mammalian digestive tract. Development. 2010;137:1721–9. 25. Furness JB. The enteric nervous system. Oxford: Wiley; 2006. 26. Furness JB, Jones C, Nurgali K, et al. Intrinsic primary afferent neurons and nerve circuits within the intestine. Prog Neurobiol. 2004;72:143–64. 27. Powley TL. Vagal input to the enteric nervous system. Gut. 2000;47 Suppl 4:iv30–2; discussion iv36.