Novel Aspects on Epilepsy by Humberto Foyaca-Sibat (Ed.)

By Humberto Foyaca-Sibat (Ed.)

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2005 Aug;26(2):63-8. N. (1998). Diagnosis and management of tuberous sclerosis complex, Sem Pediatr Neurol. , Thomas, G. R. (2006). Rapamycin causes regression of astrocytomas in tuberous sclerosis complex, Ann Neurol. 59:490-498. , Chalmers, A. M. (1987). Evidence that the gene for tuberous sclerosis is on chromosome 9, Lancet 1: 659–61. , Inoue, Y. & Fujiwara, T. (2001). Long term follow up study on Wst syndrome associated with tuberous sclerosis, Brain Dev 23:698-704. , Esterhas, A. P. (2006).

S. E. (1997). Expression of the TSC2 product tuberin and its target Rap1 in normal human tissues, Am J Pathol, 150(1):43-50. S. E. (1996). Co-localization of the TSC2 product tuberin with its target Rap1 in the golgi apparatus, Oncogene, 13(5):913-23. , Nobil, A. & Dabora SL. (2010). Comparison of three rapamycin dosing schedules in A/J Tsc2+/- mice and improved survival with angiogenesis inhibitor or asparaginase treatment in mice with subcutaneous tuberous sclerosis related tumors, Journal of Translational Medicine, 8:14.

Generalized epileptic seizures reduce total sleep time and elongate latency to REM sleep. Amount of NREM1 and NREM2 stages may be extended (Foldvary-Schaefer, 2002). Partial epileptic seizures during sleep do not disrupt night sleep markedly, only in case of their secondary generalization (Dasheiff, 2003). Epileptic seizures during daytime influence night sleep too. They reduce REM sleep, what may be cause of fatigue in postparoxysmal period (Bazil, Castro & Walczak, 2000). Antiepileptic therapy is considered to have influence on daytime vigility and quality of sleep.

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