Neuromuscular Junction Disorders: Diagnosis and Treatment by Matthew N. Meriggioli

By Matthew N. Meriggioli

This source covers all features of the prognosis and medical administration of sufferers with ailments of the neuromuscular junction. It breaks down every one disorder by means of pathophysiology, medical presentation and normal background and direction for more suitable prognosis and remedy.

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Extra resources for Neuromuscular Junction Disorders: Diagnosis and Treatment (Neurological Disease and Therapy)

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58. Matthews G. Neurotransmitter release. Annu Rev Neurosci 1996; 19:219–233. 59. Heuser JE, Reese TS. Structural changes after transmitter release at the frog neuromuscular junction. J Cell Biol 1981; 88:564–580. 60. Heuser JE, Reese TS, Dennis MJ, Jan Y, Jan L, Evans L. Synaptic vesicle exocytosis captured by quick freezing and correlated with quantal transmitter release. J Cell Biol 1979; 81:275–300. 61. Dwyer TM, Adams DJ, Hille B. The permeability of the end plate channel to organic cations in frog muscle.

64. Haimovich B, Schotland DL, Fieles WE, Barchi RL. Localization of sodium channel subtypes in rat skeletal muscle using channel-specific monoclonal antibodies. J Neurosci 1987; 7:2957–2966. 65. Ruff RL. Sodium channel current density at and away from end plates on rat fastand slow-twitch skeletal muscle fibers. Am J Physiol 1992; 262:C229–C234. 66. Lindstrom J. Acetylcholine receptors: Structure, function, synthesis destruction and antigenicity. In: Engel AG, Franzini-Armstrong C, eds. Myology, 2d ed.

The mean probability of quantal release was normal or low, and the mean MEPP frequency was not significantly different compared to controls. However, the quantal store was decreased, and the mean probability of quantal store mobilization was high. Histologic evidence of denervation and small or absent nerve terminals were observed in all patients. Only a few of these endplates were innervated by regenerating nerve fibers. Failure of electrical stimulation to generate EPPs was observed and was frequency dependent, increasing with higher rates of stimulation.

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