Musculoskeletal Disorder by M. Alricsson

By M. Alricsson

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Observe for the muscles that are under strained throughout 24 Musculoskeletal Disorder the movement. A working knowledge of biomechanics is an advantage, as it helps to identify and explain the possible pathomechanics and abnormal compensation. Evaluate the predisposing factors guided by the clinical history to locate the most probable primary trigger points and muscles implicated. Sometimes clinical skill is important to differentiate between similar symptoms caused by different pathogenesis. For instance, a middle age client with type II diabetes for 5 years with a sedentary lifestyle was recently encouraged to increase physical activity under medical advice to reduce the risk of diabetes complications.

Local muscle contractions compress sensory nerves, blood vessels and interrupt normal physiological interactions at the neuromuscular junction. The local physiological changes initiated by local ischaemia and hypoxia result in tissue damage, which releases cellular chemicals. Chemicals released by damaged tissues such as histamine, serotonin, bradykinin, prostaglandin, leukotrienes and sunbstance P, initiates the pain sequence. When chemical mediated nociceptors are stimulated, the activation threshold of a neuron is reduced and consequently the nociceptors require less stimulation to fire.

1. Simplified survey of the postural control system. Each arrow represents four tracts: excitatory- and inhibitory- on right and left side. nucl: vestibular nuclei complex; VOR: vestibulo-ocular reflex; VT: the vestibulum; III, IV, VI: occulomotor-, trochlear- and abduscens nerves; 4. Benign paroxysmal positional vertigo (BPPV) BPPV is the most frequent cause of vertigo and dizziness. e. otoliths. The cilia of the macular hair cells are embedded in this membrane and are displaced by sharing forces applied to the membrane.

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