By John M. Stern MD
Geared up by way of wave gains instead of trend names, this atlas is helping advisor the reader to an EEG interpretation even if the waveform is unexpected. the 1st part takes the reader in the course of the means of characterizing EEG waves by way of their good points. the second one part organizes EEG styles by way of their positive aspects, so offers EEG waveform differential diagnoses. The 3rd part is prepared alphabetically through development identify with every one development defined in a fashion that enables the reader to tell apart it from equally showing styles. Examples of the styles are also supplied.
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Extra resources for An Atlas of EEG Patterns
The accelerated bone resorption, which is usually the cause of the hypercalcaemia, is believed to be mediated by hormonal and/or metabolic mechanisms. The symptoms of hypercalcaemia will not be elaborated, but their severity is usually related to the level of serum calcium. 0 mmolllitre and the serum phosphate low or normal. When secondary renal damage ensues, hypokalaemic alkalosis may occur. The clinical features which help to distinguish this disorder from primary hyperparathyroidism include the rapidity of onset of symptoms and the absence of either periosteal bone resorption or nephrocalcinosis.
1 Clin Endocrinol Metab 35:764-767 Hansen M, Hansen HH, Hirsch FR (1980) Hormonal polypeptides and amine metabolites in small cell carcinoma of the lung, with special reference to stage and subtypes. Cancer 45:1432-1437 Heath DA (1976) Hypercalcaemia and malignancy. Ann Clin Biochem 13:555-560 Holdaway IM, Bloomfield GA, Ratcliffe JG, Hinson KWF, Rees GM, Rees LH (1974) Adrenocorticotrophin levels in normal and neoplastic lung tissue. TaylorS (ed) Endocrinology 1973. Heinemann, London Hope 1, Ratter S1, Estivariz FE, McLoughlin L, Lowry P J (1981) Development of a radioimmunoassay for an amino-terminal peptide of pro-opiocortin containing the 1'-MSH region.
The bioactivity of this 22-K ACTH is some 10% of 1-39 ACTH (SJ Ratter, PhD thesis). It was hoped that this 22-K ACTH could be a marker for lung tumours. However, it is also released by ectopic ACTH-secreting tumours in sites other than the lungs and recently has been shown to be released from a large and aggressive atypical pituitary tumour (Ratter et al. 1983). Since the ACTH is concomitantly synthesised with /3-LPH and y-MSH as proopiomelanocortin, it is not surprising to find that the levels of these other peptides are elevated, similarly to that of ACTH, in ectopic Cushing's syndrome (Jeffcoate et al.